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24-Hour Urine Collection (Stone Metabolic Panel)The collection that reveals why your body forms stones — volume, calcium, oxalate, citrate, uric acid, and pH across a full day. Ordered and interpreted at Remix Medical in Houston, TX.

Specialty
Nephrology
Type
Diagnostic test
CPT code
81050 (urinalysis, volume measurement, timed collection); 82340 (calcium, timed urine); 83945 (oxalate); 82507 (citrate); 84560 (uric acid, other source); 84300 (sodium, urine); 82570 (creatinine, other source); 84105 (phosphorus, urine); 83735 (magnesium); 83986 (pH, body fluid); 84540 (urea nitrogen, urine). Cystine profiles add 82131 and 84392.

Also known as: 24-Hour Urine Study, 24-Hour Urine Stone Panel, Metabolic Stone Evaluation, 24-Hour Urine Chemistry, Stone Risk Profile, Timed Urine Collection

Twenty-four hours of urine, collected into a jug, tells us the chemistry that drives your stones. Nothing else does.

A spot sample cannot. Stone formation depends on how concentrated your urine is and how much of each substance passes through it across an entire day — including the overnight hours when urine is most concentrated and crystals most readily form.

What we measure and why each one matters

Volume. The single most important number. Below two and a half liters, everything else is harder to fix. Most stone formers are chronically underhydrated and do not believe they are.

Calcium. High urinary calcium is the most common abnormality in calcium stone formers. Dietary sodium drives it, which is why salt restriction lowers urine calcium.

Oxalate. Binds calcium to form the most common stone. Raised by spinach, nuts, chocolate, and — counterintuitively — by restricting dietary calcium, which frees oxalate for absorption.

Citrate. The body's natural stone inhibitor. It binds calcium in urine so it cannot crystallize. Low citrate is common and highly treatable with potassium citrate.

Uric acid. Drives uric acid stones and promotes calcium oxalate crystallization.

pH. Persistently acidic urine forms uric acid and cystine stones. Persistently alkaline urine forms calcium phosphate and struvite stones. This single value redirects the entire strategy.

Sodium. A proxy for dietary salt, and the lever that lowers urinary calcium.

Creatinine. Not a target — a quality check. It tells us whether you actually collected the full 24 hours.

Why this is where prevention starts

Without it, prevention is guesswork. The recurrence rate after a first stone is roughly fifty percent within five to ten years. Targeted treatment based on this collection drops it substantially.

And the treatments diverge completely by mechanism. High urine calcium calls for a thiazide. Low citrate calls for potassium citrate. High uric acid with acidic urine calls for alkalinization and sometimes allopurinol. Give the wrong one and you have treated nothing.

Doing the collection correctly

Discard the first morning void. Note the time — that is your start. Collect every drop for the next twenty-four hours, including the first void of the following morning, which ends the collection.

Keep the container refrigerated or on ice. Missing a single void invalidates the entire study, and the creatinine will usually reveal that it happened.

Collect during an ordinary week. Not while traveling, not while sick, not after a holiday. We want your real chemistry, not a snapshot of an unusual few days.

Repeating it

We collect once at baseline, again several months after starting treatment to confirm the chemistry actually moved, and periodically thereafter.

That second collection is the one people skip. It is also the one that tells us whether the treatment is working, because stones take years to recur and waiting for one is a poor way to find out.

How it's performed

The patient discards the first morning void, records that time as the start, and collects all urine for the subsequent 24 hours, concluding with the first void of the following morning. The specimen is kept refrigerated or on ice throughout. The laboratory measures total volume, calcium, oxalate, citrate, uric acid, sodium, phosphorus, magnesium, pH, and creatinine. Urine creatinine serves as a validity check on collection completeness. The nephrologist interprets the profile to identify the specific lithogenic mechanism and constructs targeted therapy: thiazides for hypercalciuria, potassium citrate for hypocitraturia or acidic urine, allopurinol for hyperuricosuria, and sodium restriction to lower urinary calcium.

How to prepare

Collect during a typical week on your usual diet and fluid intake. Do not collect while traveling, while ill, during a urinary tract infection, or immediately after an acute stone episode. Certain medications and supplements may be held beforehand; do not stop anything without instruction. Keep the container refrigerated or on ice for the entire collection period.

Outcome

Identification of the specific metabolic mechanism driving stone formation, which determines treatment entirely. High urinary calcium, low citrate, high oxalate, high uric acid, and abnormal pH each require different therapy. Targeted treatment based on this profile substantially reduces the roughly fifty percent five-to-ten-year recurrence rate seen without metabolic evaluation. A repeat collection confirms the chemistry has actually changed.

Related

Learn more

Conditions treated

Your physician

Your nephrology at Remix Medical.

Every clinician at Remix Medical is board-certified and owns the practice — so the physician in your exam room is the one making decisions about your care.

  • Uday Khosla, MD

    Nephrologist

    Montrose — Upper Kirby · Limestone County — Groesbeck · Katy — Grand Parkway · East Houston — Woodforest

    Board certifiedAccepting newBook

This page is for general education and is not a substitute for medical advice. Whether a given procedure is appropriate depends on your individual evaluation. Contact a Remix Medical clinician to discuss your care.

Updated July 9, 2026.

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